QUANTUM ENGINEERING #76: HOW COME FRITZ POPP DID NOT FIGURE THIS OUT FIRST?

Fritz Popp earned a PhD in theoretical physics from the University of Mainz in 1969 and was awarded Professorship by the Senate of Marburg University. His work delved into quantum theory of many-particle systems and through his research is was able to prove the existence of “biophotons”.

WHY POPP NEVER COULD WRITE THIS BLOG SERIES EVEN THOUGH HE FOUND THE BIOPHOTONS STORY FIRST?

The reason is simple.  It was the same reason Max Plank could never explain the ultraviolet catastrophe but Einstein could.  Sir Albert looked at the thermodynamic givens, embraced their paradox, and knew nature does not make errors.  His mind went deeper into the complexity of light.  Planks guessed right that photons use quanta of light, but he stopped there.  

Popp did not understand wide band gaps because he was a theoretical physicist and not a theoretical biologist.

Popp got into biology from physics when he found out that cancer could only be linked to its optical properties and not its chemical properties.  From 1970-2018 to his death he never explained the situation.  I realized in 2005 I might have.  Let me explain.  In 1970, Fritz Popp discovered that benzo[a]pyrene, a potent carcinogen, absorbs ultraviolet light at one wavelength and emits it at another lower powered frequency of light.

He showed benzoapyrene, absorbed UV light and then re-emitted it at a different frequency (i.e. “scrambled” the light) while the latter molecule, benzoepyrene, allowed the UV light to pass through it unaltered. This told him that UV light signaling in cells was critical to get right and it required atomic precision in a cell.

The reason chemicals became carcinogenic was because their ability to enter mitosis was altered.  Popp never figured this out in all his experiments on chemicals, which is surprising.  It was clear however, he knew about Gurwitch's experiments on mitogenic radiation from the 1923 onion experiments from his writings, but I do not think he knew enough about cell cycle biology.  I think he believe the paradigm beliefs that excess mitosis caused cancer.  The real answer was that mitosis is needed to avoid cancer.  When cells are arrested at the mitosis cell cycle this is when cells are optically sensitive to oncogenesis.  

UV light frequencies are clearly needed for cells to navigate all the steps in the mitosis phase of the cell cycle.  So this should raise the question in your mind, where does this light come from?

WHAT DID POPP DO WITH THIS QUESTION?

This question led Dr. Popp to experiment with UV light and other compounds, some carcinogenic and some not. From his findings, Dr. Popp was able to predict which substances were carcinogenic by checking their specific optics - he observed that compounds that were carcinogenic would only react to light at a specific frequency (380 nm) by absorbing it and then re-emitting it at a different frequency.  In other words, carcinogenic chemicals could have identical chemical abilities but if they were "visible light scramblers" cancer would be the result.

The carcinogens seem to "scramble" the UV light signal with a wavelength of 380 nanometers. 380 nm light corresponds to a band gap of 3.25eV.  POMC responds ideally to the UV light 380 nm band gap.  380nm light also plays a huge role with mTOR biology and many other cellular processes (above).

This explained everything to me that Popp had found in his experiments.  I have not found one paper from Popp that mentioned POMC so I believe he had no idea that POMC was created from UV light in human tissues.  Without POMC there can be no melanin.  Without melanin the VUV light signals in tissues are lost and cells would be arrested at the mitosis stage of the cell cycle.  

The benign chemicals did not scramble the UV light signal but the cancerous ones did and this lowered the POMC in tissues where cancer came from while eroding their ability to make melanin.  The change in the UV signal emission caused the cells to stop their cell cycle at mitosis via optical scrambling.  

This loss of signal fidelity allowed the cells to become mobile in tissues so they would find a new source of UV light where they could then grow.  This is how wound regeneration proceeds on in Becker's experiments in salamanders.  Popp and Becker knew parts of this story, but neither of them could fully explain it.  I felt I could fully explain it because of the onion experiment of Gurwitsch and the KT event effect on melanin. There is one thing left to explain. Where do the biophotons come from?

BIOPHOTON TRANSFORMATION COMES FROM H+ LATTICE CHANGES IN THE MATRIX WITH 02 PRESENT

Time controls the flow of energy in matter.

Mitochondria are time machines who make their own ocean of water from visible light. They do not make energy, they transform it using atoms with a specific atomic arrangment in the organelle. When the organelle changes its size and shape it is a sign the time machine is a broken clock.

Energy transformation is not the sole function of mitochondria, since they have evolved as critical regulators of various cellular processes including metabolism, apoptosis, calcium buffering and cell division. The link to cell division is ANCIENT and why they should be thought more as a time machine and less as a powerhouse. Their reputation is misguided. Given that mitochondria cannot be formed de novo, it is important to gain deep insights into the molecular mechanisms governing the inheritance of preexisting organelles in each cell division in order to prevent mitochondrial damage and detrimental consequences on cell physiology. Their clock timing mechanism is critical in cell division which is a time critical event in a cell for an organism.

On Earth, in every living thing, the time of day determines the design of the mitochondrial network, and this, in turn, influences the cells’ energy capacity.”

Life uses the sun to tell cellular time. Relationship between circadian clock and energy production is not well understood by centralized medicine so I do not expect the public to understand it well either. New research shows that Life's mitochondrial network loses its key rhythms if the circadian clock is impaired, which in turn, causes a decline in energy production in the cells.

The researchers showed that the circadian clock and mitochondria interact through a protein called the dynamin-related protein 1 (DRP1), a key mediator of mitochondrial fission. Specifically, they found that pharmacologically or genetically impairing the mitochondrial Drp1 fission protein upsets the energy production rhythm, which in turn affects the rhythm of the circadian clock leading to heteroplasmy changes and disease. This is how chronic diseases begin.

Mitochondrial fission and fusion cycles are integrated with cell cycle progression. A lack of ultraweak UV light is key in this process. Inhibiting Drp1 triggers DNA replication stress, which is mediated by a hyperfused mitochondrial structure and unscheduled expression of cyclin E in the G2 phase of the cell cycle. This persistent replication stress then induces an ATM-dependent activation of the G2 to M transition cell cycle checkpoint. Cells need ultraweak UV bio-photon creation to get past the Mitosis phase in the cell cycle.

At the G1/S boundary in the cell cycle mitochondrial tubules form a highly fused network, which is associated with increased mitochondrial ATP production (PBM effect) and high levels of cyclin E, in order to promote G1-to-S transition (Mitra et al., 2009).

This hyperfused mitochondrial network is then disassembled and becomes increasingly fragmented through S, G2 and M phase of the cell cycle, with the greatest fragmentation evident during mitosis (M) in order to allow the proper partitioning of mitochondria between two daughter cells during cytokinesis.

I now believe most modern diseases result from showing a regressive evolutionary path. This is called atavism. You heard that idea in this series already in QE #45. In my opinion, most modern diseases manifest by showing evidence of semiconductive proteins undergoing photolithographic engineering inside your tissues to change light frequencies, which alters your tissues' water chemistry. This leads to new and alien bends, charges, and alteration of atoms in your tissues that change the morphology of your body and the physiology of your tissues. The most powerful changes occur in the POMC gene family and all the peptides it creates by light frequency cleavage. Changes in light frequency alter the dielectric potential in water, which sculpts semiconductive design. This is functionally how evolution occurs. It is not the path that Darwin put centralized science on.

My unconventional decentralized theory of atavism and photolithographic engineering goes against current mainstream scientific beliefs. However, It is plausible because of the science underpinning the solid-state physics that governs semiconduction and optics. While evolution plays a role in disease development, it is typically understood in centralized science via the lens of genetic mutations and natural selection = Darwinism.

The idea of light frequencies altering tissue chemistry and causing changes in the body is not well-supported by current centralized scientific evidence because no one in centralized science is allocating money to study it. BigHarma and the NIH are invested heavily in the belief that alterations of RNA and DNA are how evolution happens exclusively. They do not even allocate 1% of their funding to mtDNA studies. This shows you why decentralized action below the cell level remains hidden from the public. This blog will make you realize just how much they do not know and why we must question their authority on this topic. If you think there is no PEER reviewed literature supporting my belief that genes do not cause cancer look at the paper below from 2017.

MITOCHONDRIAL ELONGATION MIMICS GLOBE ELONGATION = DECREASED ENERGY

Thus, mitochondrial remodeling throughout the cell cycle is considered to meet the cellular energy demands during the progression of specific stages of the cell cycle, and to ensure faithful inheritance of mitochondria during cell division. However, how deficiencies in the proteins that regulate mitochondrial dynamics impact cell cycle progression and hence directly contribute to the development of diseases. Loss of Drp1 results in elongated mitochondria. Drp1 deficiency mimics what blue light does to the globe in myopia. It elongates the mitochondria. This causes mitochondrial dysfunction due to a failure of a Drp1-dependent mechanism of mitophagy that removes damaged mitochondria within the cell (Twig et al., 2008 = heteroplasmy).

When size and shape changes in mitochondria this changes the H+ lattices that are possible in the matrix. When an atomic crystalline latiice is changes they release light as a response. This idea is buried in the slide below.

The resulting accumulation of damaged mitochondria has been suggested to cause a depletion of cellular ATP and an inhibition of cell proliferation (Parone et al., 2008 TCA cycle spinning counterclockwise). Such an energy depletion-related cell proliferation defect may be caused by a metabolic checkpoint that triggers an AMPK- and p53-dependent G1/S cell cycle arrest (Jones et al., 2005; Owusu-Ansah et al., 2008). Persistent mitochondrial hyperfusion also induces centrosomal overamplification and chromosomal instability, which are causes of aneuploidy. p53 is a gene product that protects the genome, DRP1 inhibition is how you lose control of the nuclear genome. This is how DNA defects occur from mitochondrial peptide creation. = transgenerational epigenetics

KEY BLOG POINT: HOW BIOPHOTONS ARE MADE

Mitotic machinery transforms energy from matter (matrix H+ lattice changes from elongation) using oxygen and H+ to stimulate light release in the form of biophotons, which in turn, to regulate mitochondrial homeostasis.

Why is oxygen needed to make biophotons? You won't find this answer in Roeland van Wijk's book on this topic. You will find it here for 5 bucks. Oxygen is a powerful element for the human gut because of what it does to electrons. Oxygen use by life began the initial penetration of solving complexity in tissues and organizational structure. It being the only paramagnetic gas on the periodic table makes it pathway unique for information processing in a quantum cell. O2 has two unpaired electrons. Electrons are key parts of mass to run photons in tissues: see the photoelectric effect.

Tissue complexity is enabled by the number of electrons, and is responsible for producing more electrons in a thermodynamic system. This is why life exploded after the Cambrian event. There is no other reason. We need more electrons to get more oxygen so we can generate more electrons to carry light in the system. This is why humans lost their fur. Their skin became a new mode to recover more electrons from the sun, in the from of photons. Your skin is a solar panel for the complexity in your chest and brain.

Oxygen's electrons are really more important to how humans work in decentralized fashion. For Example, if you have any skin disease (neuroectoderm derivative) that is associated with an altered immune response, do you know why the sun is your Rx for wellness? Did you know singlet oxygen is a potent trigger for the induction of human T cell apoptosis because of its electrons. Did you know UVA light from the sun is the most potent trigger to singlet oxygen production in the all neuroectodermal derivatives? Did you know melanin in the skin augments this effect because it is also a neuroectodermal tissue? I doubt you do because no centralized MD does.

Remember electrons and photons are basically the same thing with respect to how the photoelectric effect operates in the quantum realm (oversimplified obviously). They are the particle in Nature which has a way to capture a massless source of energy and information contained in light and move it by ionization or delocalization. This helps explains why all cells release ultraweak UV light. You should also realize that UV light creates oxygen in the atmosphere and in the venous side of your circulatory system. Big implications for chronic diseases when hypoxia is present in the system; it causes things to go awry.

Any replication stress then initiates the DNA damage response.

Proof: All living cells emit ELF-UV light and how we see how fluids and atoms are moved in cells = AMO physics 101.
https://www.eurekalert.org/news-releases/518737

More sunlight = more information in the system to build complexity = more oxygen
Time appears as entropy goes from order to disorder. When entropy is controlled by a dissipative system, time can appear to be illusory.
This means that the less information you have about a data set in a cell, the higher its entropy must be in the system. Cells limit entropy by controlling their atomic arrangements. In precise terms, entropy is a measure of the number of possible atomic arrangements that a system of particles can be in.This is important in understanding how Nature engineered our biological clock gene to work.

Remember that stored energy in HEALTHY cells is coherent energy. The organism is, therefore, a highly coherent domain possessing a full range of coherence times and coherence volumes of energy storage. This keeps it far from equilibrium and makes it a highly dissipative system of organization to control entropy.

Reminder: Cellular organization is the key to precision optical signaling. Life is all about optimizing AMO physics INSIDE OF CELLS. It transforms energy from the environment to do this. Modern physics now has proven that energy and information are equivalent in physics. Landauer's Principle of 1961 & Shannon's 1948 work was critical in making this linkage. Modern quantum biology has experimentally proven that energy is trapped directly at the electronic level in cells. Energy is stored not only as vibrational and electronic bond energies in biochemicals, but also in the structure of the system: its enzyme kinetics, membranes, and in gradients, fields and flow patterns, compartments, organelles, cell water, and tissues. All this in turn enables organisms to mobilize their energies coherently at any time it is needed and hence make available the entire spectrum of stored energies for physiological work. It is energy on demand by atomic design.

SUMMARY

During his work with chemicals, Popp learned that 380 nanometers, the wavelength altered by carcinogens, is also the key wavelength that cells prefer to use to repair themselves. After exposure to intense UV light, cells quickly self-repaired themselves when they are exposed to very weak UV light, particularly that with a wavelength of 380 nanometers. Popp hypothesized that cancer results from a disruption of cells' photo repair system. What he never seemed to realize was that 380nm is the frequency of the mitogenic radiation in Gurwitch's onion experiment that restored mitosis in cells.

His hypothesis raised a question: what in the body produced this very weak light that powered the repair system? Popp and his student Bernard Ruth found that all living systems store light energy (photons) acquired from the sun and from plants consumed as food (photosynthesis), in DNA. This stored light is released as very weak, extremely coherent biophotons. UV light photons made from wide-band gapped semiconductors switch on the body's processes like a conductor launching each individual instrument inside the cell.  This orchestration is how the products are cleaved from POMC in different tissues to lead to disease or wellness. It was the alteration of the band gap that was the key to optical control in a cell that led to "the collective sound" at different frequencies as they perform different functions.

Over the years, Popp found that biophoton emissions from healthy humans display rhythmic patterns. He never realized those patterns linked to circadian changes of the hydrogen bonding networks in water.  He also observed that the coherence of the light emissions, the intensity, and the rhythmic patterns varied in people with different illnesses. 

For example, people with multiple sclerosis absorb too much of the wrong light and their photon emissions display too much order and this affects the opening and closing of the AQA 4 gates in the CNS/PNS.  

The change in frequency of light changes oxygen tensions in the cell and mitochondria and this changes the ROS/RNS signals and the light emission of the cell.  When the light emission is changed, migration of the glial cells can cause AQA4 gate malfunction.  

This can be mediated by the divalent atoms in the mitochondria of those semiconductive gates.  Moreover, they stop working properly moving water with each action potential.  nnEMF can change the band gap of a system in a cell just by changing the VGCC on the membrane with a specific frequency.  (see below)

The sun creates a different calcium signal in MS patients.  The paper above shows that nnEMF also carries the ability to change calcium and magnesium flows in mitochondria and this will alter the band gap in these organelles.  This will change the free radical signals, oxygen levels in mitochondria and ultimate change the light emission from cells.  If light emission is changed  in glial cells Multiple Sclerosis is the likely outcome if glial cells migrate away from the AQA4 gate.

Schwann cell precursors (SCPs) are glial progenitors, closely associated with developing nerves of the peripheral nervous system along which they migrate, sometimes long distances, throughout the body. SCPs are derived from neural crest cells (all contain melanin) that emigrate from the neural tube and migrate into the periphery. Accordingly, SCPs closely resemble neural crest stem cells but also have properties that are characteristic of immature Schwann cells.   In our adult form Schwan cells appear to have migratory ability like melanocytes and WBCs in our immune system.  I believe MS is an abnormal migration of neurons due to an altered VGCC's in their mitochondria that leads to short circuits in the neurons.  

ALS MIGHT ALSO BE A MIGRATORY NEURON DISEASES DUE TO A LOSS OF ENDOGENOUS UV SIGNALING

All spinal motor neurons derive from motor neuron progenitor cells, located in a restricted ventral region of the developing spinal cord.

During late gastrulation and neurulation, the developing spinal cord is called the neural tube, and is patterned into distinct progenitor domains. MNs are specified from progenitors in the ventral neural tube. Once specified, newly born MNs are further specified into columns, pools, and subtypes, forming a unique topography. From these columns and pools, axons reach out to their targets under varying guidance cues. All MNs are cholinergic cells which integrate with the motor control circuit, the sensory system, and their outlying targets to control movement.  Given the growing importance of the MN–glia interaction in a number of neurodegenerative diseases it is important to know that the initial specification of oligodendrocyte precursor cells (OPCs) share a common progenitor with MNs.  This implies that motor neurons could degenerate into oligodendroglia or even back into neuroepithelium where they come from in the embryo.  

Motor neurons (MNs) are neurons located in the central nervous system (CNS) controlling a variety of downstream targets in muscles. There are two main types of MNs, (i) upper MNs that originate from the cerebral cortex and (ii) lower MNs that are located in the brainstem and spinal cord.  This explains why some forms of ALS is worse than others.  

If the nnEMF changes anterior and posterior neural plate neuron migration signals might explain why ALS occurs when anterior motor horn cells disappear.   The anterior end of the neural tube will develop into the brain, and the posterior portion will become the spinal cord. The neural crest develops into peripheral structures. At this point, the early nervous system is a simple, hollow tube. It runs from the anterior end of the embryo to the posterior end.

In vertebrates, neuroepithelial cells give rise to the neural tube, which forms through two processes along the anterior-posterior (AP) axis. The first process is primary neurulation, which progresses through convergent extension, elevation, bending, and fusion of the neural plate, forming the rostral neural tube. By the end of primary neurulation, only the brain and anterior trunk structures of the spinal cord have formed. As the embryo develops, progressive addition of new neural progenitors (NPCs) is required at the posterior end of the spinal neural tube for neural tube elongation. The cells at the dorsal region of the tail bud aggregate and the tail bud ultimately undergoes cavitation, forming the caudal neural tube; this comprises the future caudal domain of the spinal cord, which is in continuity with the neural tube in the trunk derived from the primary neurulation

Amyotrophic lateral sclerosis (ALS), also known as motor neuron disease (MND) or Lou Gehrig's disease, is a neurodegenerative disease that results in the progressive loss of motor neurons that control voluntary muscles in humans

Currently no one knows where these anterior horn cells go, but given the papers on Zebrafish out of Stonybrook University in New York I bet the motor horn cells have altered melanin biology in those cells and this leads to their migration somewhere else in the nervous system.  This would mimic what we see in melanosomes mentioned in Quantum Engineering #30.    

Why do I say this?  

Human bodies, like those of other vertebrates, form in a ‘head-to-tail’ direction during embryonic development. There is growing evidence that this process is fuelled in large part by a pool of proliferating cells called neuromesodermal progenitors (NMPs; reviewed in Henrique et al., 2015). These cells have been found in zebrafish, chick, mouse embryos, and in human embryos (Olivera-Martinez et al., 2012).  

Moreover, they seem to produce both the neural tissue that makes the spinal cord and mesodermal tissues such as muscle and bone. 

Vertebrate embryos establish their primary body axis in a conserved progressive fashion from the anterior to the posterior. ALS is a disease that is linked only to anterior motor horn cells.  During this process, a posteriorly localized neuromesodermal cell population called neuromesodermal progenitors (NMps) plays a critical role in contributing new cells to the spinal cord and mesoderm as the embryo elongates. Defects in neuromesodermal population development can cause severe disruptions to the formation of the body posterior to the head. Given their importance during development and their potential, some of which has already been realized, for revealing new methods of in vitro tissue generation, there is great interest in better understanding NMp biology.

The nervous system of vertebrates can be understood as a means of internal interconnection that enables multicellular animals to coordinate their different physiological activities and interact with their environment.  ALS presents a seeming paradox to centralized healthcare because they do not understand how light sculpts tissues. 

Zebrafish research at Stonybrook University showed  rhythmic patterns. Also, tumors emit high amounts of photons: an average of 300 [+ or -] 90 photons/cm per minute compared with normal tissue emits an average of 22 [+ or -] 6 photons/cm per minute.  Human cells that emit too much light seem to be a problem.  I do not currently believe the amount is the issue I believe the frequency is the problem and this stops the cell cycle in mitosis.  When this occurs oncogenesis begins.

Popp and colleagues at the International Institute of Biophysics discovered that surface tumors and tumors excised during surgery respond to remedies with changes in photon emissions. This helped me understand mammal metastasis at the KT event and what the real cause of melanoma is today.

CITES

https://www.cell.com/cell-metabolism/fulltext/S1550-4131(18)30063-9

https://journals.lww.com/oncology-times/fulltext/2019/01050/non_proliferative_cancer_cells___the_deadly_charge.5.aspx

https://elifesciences.org/articles/14830

Elisabeth Zieger, Michael Schubert. New Insights Into the Roles of Retinoic Acid Signaling in Nervous System Development and the Establishment of Neurotransmitter Systems. Int Rev Cell Mol Biol, pp.1-84, 2017. hal-02117372

https://threadreaderapp.com/thread/1636019966947348480.html

https://x.com/DrJackKruse/status/1795236535069008254

Hydrogen = H+. Deuterium = D. . It is the atomic chameleon. It is the first element on the periodic table. It is what our sun is mostly made from and burns most to make energy. It is the element found in greatest density inside of a mitochondria. This makes it an interesting study point for Einstein's relativity and cosmology and QED. Mitochondria have a really small scale of action and this small-scale effects the idea of relativity which is based upon geometry. Without the scale of geometry gravity's effect is lessened to a great degree. Hydrogen (H+) has the ability to be a metal when it loses its electron; this is what happens inside our mitochondrial matrix. As H+ is can act a superconductor. Superconductors have special crystalline lattice and when that lattice is deformed it releases light. This makes it the focus of quantum electrodynamic theory. This means our mitochondria is filled with ionized plasma. This is very similar to what the sun does when it burns hydrogen. Hydrogen can be non metal when it has its electron orbiting its sole proton; It is also can be an acid because H+ is the basis of pH scale which measures acidity. Deuterium and a proton, have separate abilities and chemistry that varies. They both show up differently in MRI scans because of the differences in their physics. H+ and D interacts with water differently. H+ has the ability to do some unexpected things because of its ability to proton tunnel. The barrier for deuterium to tunnel is markedly reduced compared to H+. When we add infrared light H+ really become quite special. With 1538.5 nm photon frequency added to cell water, proton transfers in water are increased dramatically. This is called proton tunneling. This effect changes receptor biology and enzymes. Did you know every biologic enzyme known to man uses proton tunneling to work? All DNA and RNA only work when they are hydrated because they require hydrogen protons to tunnel!!! This makes hydrogen life's magic weapon in enzyme and receptor actions. It has the same effect on DNA and RNA as well. Enzymes are 100% quantum experiments in energy and information transmutation. So how does hydrogen fit both Relativity and QED theory? Read on..........................https://nautil.us/will-quantum-mechanics-swallow-relativity-235658/

The living universe selects for maximum entropy, and minimum waste heat.

The implications of this idea include:

The emergence of complexity: The universe's drive for maximum entropy and minimum waste heat led to the melanin renovation Rx. This led to the emergence of complex structures and patterns, for life on Earth. Complexity came from becoming able to using the TCA in counterclockwise wise spin (anaerobic old system) and the clockwise wise spin (aerobic new oxygen Earth) during the same lifespan. New software patterns, like moving melanin to the mammalian interiors to begin to use endogenosus melanin to control the spin cycle of the TCA had to be innovated.

The arrow of time: The universe's tendency towards increasing entropy explains why the human brain uses dopamine and melatonin as clock gear proxies. Both are critical in renovating all non visual photoreceptors in us. There use is why we perceive time as moving in a particular direction. Mammals' criticality begins with UV light because it works with mtDNA, quantum dots, and melanin to create VUV-IR light inside a cell. When the critical amount of these entities goes missing inside mammals, they suffer a loss of healthspan or a loss of time.

The nature of consciousness: The self-organizing nature of the universe has massive implications for our understanding of consciousness and the human experience. The nature of consciousness is buried in the queerness of water's abilities and what ultraweak biophoton spectral frequency change can do to steer water in its holographic format. Scientists at the US Department of Energy Oak Ridge National Laboratory (ORNL) have discovered new properties of water that go beyond the known laws of classical physics.

https://x.com/DrJackKruse/status/1776818496032153937