QUANTUM ENGINEERING #36: THE SUN DOES NOT CAUSE SKIN CANCER, A LACK OF SUN DOES

Finsen figured out long ago the light in the sun was the key to healing many diseases.  In 1893 he showed IR-A light cured smallpox.  No vaccine was needed.

Kellogg, or cereal fame, followed this science and found that the sun truly was miraculous.  Since the 1890's centralized medicine has tried to demonize the sun & Finsen's work.  Kellogg knew back at the end of the 19th century that part of the sun was good for us he and his rich friends decided to bury that truth to build an industry around food and drugs to take advantage of this knowledge.

The paper below is over 20 years old.  It says that with repeated UV exposure skin with more melanin is associated with FASTER DNA repair.    This is 180 degrees to the dermatology opinion that the sun is toxic.  This says the more melanin you have in your skin the faster you heal.

Note the color of type 2 skin to type four skin that was mentioned in the paper.  Do you notice that darker skin is a huge advantage?  Most African Americans fall into types 5 and 6 six types and many of them still get sunburn when they go out in the sun as modern humans. Do you know what sunburn means in their case?

Did you know that skin cancer rates in Type 5 & 6 skin are reduced?  Can you venture a guess why now?

People of color have a lower risk of developing skin cancer than people with fair skin tones.  Imagine that.

The paper below shows avoidance of the sun is a risk for death on a par with cigarette smoking.  You cannot make this stuff up.

OVERVIEW OF GURWITSCH'S 1923 ONION ROOT EXPERIMENT FOR REVIEW

To test the hypothesis of the “non-chemical external impulse,” Gurwitsch performed his famous “onion root experiment”. Two onion roots as even and smooth as possible were located perpendicular to each other and mutually centered, so that the tip of root No1 (acting as the “emitter” of the “impulse”) was directed toward the division zone of root No 2 (acting as the “recipient”). The authors made histological sections of the “recipient” root, and calculated the number of mitotic figures in the exposed and non-exposed halves of the root. The exposed side possessed significantly higher proportion of cells in mitosis than the non-exposed side. This phenomenon was called “mitogenetic effect” (MGE).

MGE was also detected, if a quartz plate was fixed between the two roots, and was not detected, if the roots were separated with glass or nontransparent materials ( confirmed by Gurwitsch, 1924; Reiter and Gabor, 1928a). Chemical isolation of the roots did not affect the results. Based on these and other data, the acting factor was concluded to be UV light of very low intensity, and was called “mitogenetic radiation.”

Back to the blog.................

What are the skin and eye doctors missing?  Neither one knows about Gurwitsch's work on mitosis and UV light, neither know that cells that can't divide travel in our body to look for UV light release, and fewer have realized that modern lighting has totally subtracted out all UV light and most of the IR-A light leaving behind mostly blue light which causes massive ACTH release which drives blood glucose and insulin levels through the roof while also causing melanin degradation on our skin.  High blood glucose and insulin make the melanin in your skin atrophy and the lack of environmental UV light allows melanocytes to become mobile in the neuroectoderm migration patterns.  This means you cannot absorb UV light well via your skin even if you were on the equator naked.

Why?

Answer:https://twitter.com/DrJackKruse/status/1636019966947348480

As result what happens? If you cannot absorb UV light because your melanin sheets are disrupted or dysfunctional due to a lack of UV-IR-A light on your surfaces,  the melanosomes deep inside your skull and viscera begin to migrate toward your surface to find UV light at the surface.  Here is where Einstein's relativity bites you in the ass.  Without UV light created endogenously, you lose the ability to slow electron flow on the inner mitochondrial membrane.  This is why Mother Nature put the VDR receptor there.

When this occurs in mitochondria, we lose the ability to create melatonin in mitochondria deep inside our body, and slowly over time apoptosis becomes defective.  Mitochondria and immune cells take our defective engines.  Without endogenous VUV production, these organelles and cells cannot work well.  What are the acute symptoms we see in our patients that this is ongoing slowly?  Women tend to get melasma and hypothyroidism.  Melanosomes migrate to the surface of their faces from deep in their skulls because of the surface's use of makeup, sunglasses, and sunscreen.  A lack of UV light and extreme use of blue light drive this process.  This is why I wrote this blog below long ago.

Cancer states all have one thing in common and it links directly back to melanin biology:  Apoptosis physiologically doesn't operate as designed because the VDR gets redacted on the inner mitochondrial membrane.  You can finally understand the Warburg effect when you understand how POMC biology and a lack of UV light are driving this process. Cancer cells have to keep bringing electrons to ECT, and this process happens because of ACTH from POMC.  65 million years ago this allowed mammals to survive without food.  Today, because UV light has been deleted for longer time periods in humans than it was 65 million years ago, new collateral effects have occurred.  A chronic cleavage of POMC - high levels of ACTH release to drive glucose and insulin levels.  POMc mimics what photosynthesis does, it creates glucose directly from light.  Mammals use blue light frequencies to cleave ACTH directly from POMc to do it when UV light is absent from the environment of the mammal.

This tells us that endogenous UV light has to be liberated in an uncontrolled fashion when this process is present in mitochondria to create a constant source of blood glucose and insulin to maintain the pace.  We know from Pritz Popp's work when eukaryotic cells have defective mitochondria they emit more UV light.  They are designed not to release light.  Prokaryotes release 5000 times more light than eukaryotic cells by design.  This is a big clue we are using non-linear optics to signal.  It is the control arm of all biochemistry.  65 million years ago this mechanism saved mammals with a disrupted food chain on Earth.  This implies that the acute state of a lack of UV light control helps mammals survive for short periods of time. This situation told me the sun was not disrupted very long because if it was, mammals all would have died from cancer and they clearly did not.  On a chronic basis, the lack of UV light controls on POMC will drive cancer diagnosis and growth.  That is the modern burden of mammals today because this subtraction has gone on since 1893.

Blue light and nnEMF drive this process due to the products in POMC and how they operate with melanin and melatonin production.  I hinted at this in a big way in the Time 9 blog.

A lack of UV light creates chronic diseases in the mammalian system because of POMC biology. Most of what we call today's mammalian chronic diseases really are just adaptations built into our cells.  The chronic maladaptation of light builds a facade that we call diseases.  A lack of UV light drives insulin, blood glucose, and precocious puberty on an acute basis.  Chronic UV blockade means mammalian cells on their skin cannot get into mitosis.  This makes the grow due to the insulin and growth signals and allows them to migrate.  This is why cancer is exploding in mammals in the 20-21st century.

Only electrons can capture photons.  So what makes cancer worse?  A lack of UV light or a lack of electrons in key spots is a real problem.  Another issue could be too many protons in water, which changes its dielectric constant and refractive index.  This means we cannot absorb enough UV light in water.  These biophysical factors are present in our blood, if you know what to look for, those changes will be in cells bathing in this water.  Once a clinician sees the cellular changes they know by definition what is happening on the quantum levels in hydrogen bonds in water: namely the thickness of the coherent domains in water made inside a cell from mitochondria.

This is why every patient who hires me at my clinic gets a peripheral blood smear.

This may sound tough to figure out until you realize what POMC is really doing.  Once this perspective is in your head, you can never go back to a centralized medicine mindset.  It is a game changer at the larger scales of practice. It fully explains why the Warburg shift works with glucose and glutamine to maintain ECT function while inhibiting apoptosis because UVA and UVB light cannot stop ECT flow before the ATPase.  It is wise for the mitochondriac to remember that the first step in heme synthesis also begins in the mitochondrial matrix.  So it is affected, so will your RBC and hemoglobin.  If they are the melanin renovation Rx will not work.

Anytime intracellular water production is lowered from mitochondrial respiration or physically changed in ways below your perception, proteins are less hydrated and this affects the physical chemistry of divalent ions like calcium and magnesium that work with mitochondria in stressful situations. The velocity and chemical activity of ions is determined by the degree of their hydration and the atomic mass of things in that WATER. This is why dehydration is devastating to cellular metabolism in the cytosol and in the mitochondria.  Both of these ions are paramagnetic and drawn to organelles with inherent magnetic fields.  When mitochondria are stressed their ATPase cannot spin as fast and as such their rotating heads spin less fast.  As a result, these local mitochondria begin to sense time differently, and that effect is seen in the size and shape changes in mitochondria, the formation of IMJ (pic above), and the space between cytochromes.  These are the telltale signs of cellular information loss.

MELANIN RENOVATION Rx

It begins with AM sunlight because this light is loaded with IR-A light.  This pre-conditions your skin to absorb more UV light at the transition at your particular latitude. (more on this topic later in the series)

The effectiveness of UV to induce erythema declines rapidly with longer wavelengths as we get closer to 400nm. To produce the same erythemal response, approximately 1000 times more UVA dose is needed compared with UVB. UVB-induced erythema occurs approximately 4 hr after exposure, peaks around 8 to 24 hr, and fades over a day or so; in fair-skinned and older individuals, UVB erythema may be persistent, sometimes lasting for weeks. The time courses for UVA-induced erythema and tanning are biphasic. Erythema is often evidenced immediately at the end of the irradiation period; it fades in several hr, followed by delayed erythema starting at 6 hr and reaching its peak at 24 hr. Erythema is associated with a wide variety of changes at the cell and molecular levels, but especially with the appearance of apoptotic keratinocytes (sunburn cells). The action spectrum for UV-induced tanning and erythema are almost identical, but UVA is more efficient in inducing tanning whereas UVB is more efficient in inducing erythema. The observation that the action spectrum for erythema is very similar to that for CPD induction suggests that DNA damage is an important trigger for erythema.  You will be shocked to find out that DNA damage induces ROS/RNS that melanin absorbs to charge separate water to liberate 2 electrons.  This mimics the first step in photosynthesis in plants.  This means ROS/RNS are GOOD THINGS, and they are only bad things when melanin is absent.

The story in the literature is filled with papers to fight against the dermatologist's opinions.  Sunburns won't give you skin cancer and they certainly not kill you.  They actually may be life-saving as the paper below shows from 1980.

SUNBURNS DO NOT MATTER. Another bad meme people spread is because they are ignorant of the effect of melanin from UV light exposure via POMC.

And the flip side of this argument is sun exposure actually leads to an all-cause drop in mortality. Burn away. I do and I'll never change that opinion because I am a mammal and I understand my clades genesis and why we flourished when UV went missing.  
****All-cause mortality: http://ar.iiarjournals.org/content/38/2/1173.full

Best time to put your Junk in the sun?

UV-B light is the off switch for sex steroids as laid out in the Vermont talk and talked about in blogs. UV-B light inactivates sex steroid creation from sterols to maximize Vitamin D production.

CRITICAL Brain-Gut POINT ALERT: When this chemical effect is CHRONICALLY present, the decision in the cell always has to be made between survival or reproduction based upon how the cell signals using its nuclear hormones.  When we are oxidized we are consuming our hormones. UV light actually inactivates our sex steroid hormones. This is another form of natural childbirth in the summer months when UV light dominates.

When we are reduced we are resupplying them in the great pharmacy in our brains by using melanin to restore pituitary POMC hormones. This means that all the LDL cholesterol that is normally made into pregnenolone will either go into cortisol OR to the progesterone pathway. If all the pregnenolone shunts cortisol’s path, it helps you survive life’s oxidation. The shunting signal that determines that choice is the level of cellular inflammation that oxidizes the cell.

Lipid POINT:  Sunlight reduces cholesterol, LDL, Lpa, and APO B too.  No drugs are needed.  Eumelanin is used to step down UV power to create a small amperage ferroelectric current that lines the surfaces of all mammals.  When that current is interrupted this is where you will see lipid accumulation, endothelial disruption, lack of NO production, and damage to melanopsin chromophores.  POMC will also be destroyed.

When we measure cortisol in the plasma, saliva, or urine, it is often low when we are oxidized chronically. That is a sign the PVN nucleus in our brain is working overtime because melanin there needs renovation, and this is a sign you are oxidizing your cells. You are aging faster than normal.

UV light exposure is the key to reducing a tissue in mammals because electrons are moved into the tissue and protons are moved out and the pH change improves the VUV light power made in the tissues. This stimulates POMC production while renovating the melanin in your cells to regenerate from stem cell depots.  The movement of electrons occurs by inducing Becker's injury current using ferroelectricity.  The same mechanisms work in the adrenal medulla as well.

This is measured clinically by an adrenal stress index test and really accurate in a low salivary melatonin level and flatlined cortisol curve. The result is all the hormones going the “other way” in the hormone synthesis chain are very low……..that is the “reduction path”. Reduction means you are staying younger because melanin renovation is progressing.

This explains why human babies are supposed to be born during the late spring and summer.  Nature uses sunlight to lower the father's testosterone and this fits nature's plans on many levels in the family unit.

Sunlight increases melanin by way of POMC cleavage of the endogenous release of cellular UV light, Vitamin D3, histamine, and sulfhydryl groups while lowering (photolysis) adrenalin, steroids, testosterone, estrogen, thyroid hormone, DNA, and RNA. This is the feedback loop.  Other chemical liberated adjacent to POMC acts to lower the sex steroid hormones.

Secretion of hormones by the anterior pituitary gland can be stimulated or inhibited by paracrine factors that are produced during solar reactions.  While UV stimulates all the things in POMC including ACTH, the product of ACTH is ultimately glucose and glucose provides feedback control to decrease POMC signaling and this stops melanin production via alpha MSH.  The elevated blood glucose continues due to this feedback loop allowing all mammals to survive long periods of time without food.  This is the pathway mammals use to hibernate.  It turns out that elevated glucose acts like antifreeze in mammals and keeps their blood from freezing and it also stimulates another subpopulation of neurons that link to reproduction fitness and thermoregulation.

Kisspeptin, Neurokinin B, and Dynorphin regulate reproduction.

KNDy neurons are located in the hypothalamus region of human brains due to conservation across ALL mammalian species. Other roles of KNDy neurons include influences on prolactin production; puberty; stress' effects on reproduction; and the control of thermoregulation.

The mature male testis has two primary functions: sex steroid hormone production and spermatogenesis both of which are needed for mammalian survival

The roles of testosterone and 5-alpha-dihydrotestosterone (DHT) in male sexual differentiation are not germane to this blog.

THE HYPOTHALAMIC-PITUITARY-TESTICULAR AXIS

This axis is controlled by a classic feedback loop. The major endocrine stimulators of human testes are luteinizing hormone (LH) and follicle-stimulating hormone (FSH), which are made by the anterior pituitary via light coming through and energizing the central retinal pathways and secreted into the systemic circulation. LH stimulates the testicular synthesis of testosterone and its two major active metabolites, estradiol and 5-alpha-dihydrotestosterone (DHT). LH is secreted in a pulsatile pattern with peaks approximately every 90 to 120 minutes. FSH has a subtler pattern of pulsatility. LH and FSH secretion is stimulated by the pulsatile release of gonadotropin-releasing hormone (GnRH) from neurons in the hypothalamus; GnRH reaches the gonadotroph cells of the anterior pituitary via a portal vascular system.

The hypothalamus GnRH pulse generator reacts to light — The medial basal region of the hypothalamus (particularly the arcuate nucleus) contains neurons that secrete a gonadotropin-releasing hormone (GnRH) from axon terminals in the median eminence (has no BBB) into the hypothalamic-pituitary portal system. These neurons constitute the GnRH pulse generator and act as the metronome of the axis. Because serum concentrations of GnRH in the portal system are normally low, peripheral circulating GnRH concentrations are very low and not measurable in humans. Several hormones, neuropeptides, neurotransmitters, and cytokines modulate GnRH secretion.

Kisspeptin and its hypothalamic receptor, KISS1R (formerly called GPR54), play a major role in stimulating GnRH secretion, and it is likely that a synchronized interaction between the secretion of kisspeptin and the coexpressed neuropeptides, neurokinin B and dynorphin (from KNDy neurons of the arcuate nucleus), regulate the pulsatility of GnRH secretion. A number of neurotransmitters and hormones regulate GnRH secretion, including gamma-aminobutyric acid (GABA), glutamate, and leptin (stimulatory) and sex steroid hormones, corticosteroids, and opioids (inhibitory).

Kp does not work alone.  DYN A is an endogenous opioid that inhibits GnRH.

Apart from the well-established role of kisspeptin (Kp) in the regulation of reproductive functions, recent data described its action in the control of metabolism. Of particular interest for the review is the population of Kp neurons localized in the arcuate nucleus (ARC) of the hypothalamus, the site of the brain where reproductive and metabolic cross-talk occurs.

Sunlight induces biochemical reactions via photolysis and it induces coordinated endocrine adaptation effects in the eye and the skin surfaces where melanin and leptin dominate in mammalian physiology. It affects the sympathetic and parasympathetic systems where POMC dominates in these neurons.

It is the stimulus for the circadian timing mechanism of the body clock via the central retinal pathways. All these effects are built into the electronic state of your semiconductive proteins under solar power and magnetic flux.

If you re-read Brain Gut 11 you will see what a chronic low cortisol level buys us. Poor solar exposure chronically = chronic Low cortisol = low mitochondrial melatonin = epithelial cancers = Leptin Resistance = a lack of melanin in that tissue. These are the chronic effects.

Acute light stress with blue light will stimulate glucose and insulin production and precocious puberty in mammals.  Mammals used this on an acute basis to survive the interruption of sunlight and still procreate.

We tend to get cancer as we age. It follows then that oxidation = Leptin Resistance and LR = aging. Low cortisol is not a good thing for a human long term, but short term it could be adaptive.

When the process first begins……ACUTELY, you will have hypercortisolism for a time, until you fatigue the output of your PVN nucleus in the hypothalamus. That PVN nucleus is just one of the major pharmacies that function in your brain. Oxidation occurs when you cannot use the TCA or urea cycle optimally. If you do that long enough, you oxidize (age) your body, while simultaneously destroying your sleep, causing your body to slowly begin to fail while your body composition declines.

For example, Hashimoto’s disease is a disease of chronic oxidation with melanin degradation throughout the human nervous system. It depletes you of the life-giving chemicals in the pharmacy that resides in your brain. This is why it is associated with so many other neolithic diseases.  These are all due to a lack of melanin where T3 and T4 are made.  The slide below shows you melanin can be used to restore thyroid hormones.  These are reversible reactions in all mammals.

SUMMARY

Realize that the number of genes an organism has in its genome is linked to the amount of energy the organism can transform.  This means the gene expression is also directly correlated by probabilities to how it is expressed.  POMC in mammals is critical in this energy linkage.  This is a function of the energy/information flow from their environment to their skin/eyes/guts, and not the anatomy of the genome itself.  It is also related to the redox potential of the organism in question.  Ultimately, All energy in life ultimately comes from sunlight.  It is stored in every cell membrane and in the electronic state of cells.  Most of these stores of energy available to cells are not accounted for in any biochemistry book.  Get some sun today to give your genome the day off to rest once in a while.  Your health will benefit if you do.

The POMC system was built in animals before the age of mammals 210 million years ago but mammals refined and now define this ancient light system (controls their entire epigenome) and brought it to prominence in their clade when dinosaurs were dying due to an asteroid collision.

The change in light frequency from this impact is what sculpted mammals to amplify this system.  The POMC system in the mammalian skin and fur was key to their survival.  From this point in their history, they amplified POMC in all neuroectodermal derivatives and this meant they did not need more genes to advance from an evolutionary perspective as all other animals had in the past.

Fast forward 65 million years, and now humans are the trophy "mammal" on Earth.  They are at the top of the food chain of all mammals because of how they have used the POMC system to sculpt their neuroectoderm compared to any other member of the family.  This idea explains a paradox from the human genome project as well.

Scientists were shocked to find humans and primates are almost genetic equivalents.  This goes from primitive primates to humans.  POMC biology explains fully why humans (mammals) have virtually the same number of genes as their recent ancestors.  In the last 2-4 million years homo-sapiens primates have used melanin to sculpt their nervous system using a light-saber from the heavens.  Melanin biology links directly to mitochondrial redox power and this is why the energy power laws still exist in mammals.

At this point, embracing technology and nnEMF is like booking a ticket on the Titanic or getting in the car with Thelma and Louise.   The reality is this: As a result of your awesome internet connection, you'll get an awesome connection with the hospital too!

Unless humans can make their original adaptations to our environment as rapidly as their science can alter Earth, our culture and society will continue to drive our species to extinction.  The development of a bio-physics understanding of mammalian biology is a critical prescription for the protection of the species requiring the experimental spirit of the modernist vanguard.