
Humans are not broken, but the environment they have built is ruining their health. You must realize that air pollution to your skin is like wearing sunglasses to your eyes in how it reduces the quantum yield of sunlight to your arterial system.
Here is the link that shows you this is not hyperbole.
https://news.byu.edu/news/byu-researcher-links-air-pollution-blood-vessel-damage
I hope you begin to realize that hemoglobin and the arterial tree are designed to have the same spectral frequencies to red light in sunlight. Recall red light makes up 43% of sunlight. That is the largest fraction in sunlight. So this begs the question what happens when the optical density or the charge density of hemoglobin and the arterial walls change with respect to each other when the sun is blocked by something? We call this change a change in the optical density of tissues. This fundamentally changes how both tissues can interact with sunlight just below the skin's surface. That is where your arteries are located.

This relationship is very similar to how a leaf controls the photosynthetic capacity in plants by altering the optics of light.

When the optical density of arteries change in relation to hemoglobin, we can observe a frequency shift in the arterial wall and this leads to a faster ubiquitin marking in the vessel and it ages faster in how proteins are altered as light frequencies change. As it ages its interaction with light changes by inducing a frequency shift in the light it will interact with. The result is a lack of cholesterol sulfate in the vessel wall, in the plasma of blood and in the collagen of the skin. What occurs when light undergoes a frequency shift? Mitochondria make less melatonin and water.

Might atherosclerosis best be explained as being due to cholesterol sulfate deficiency in blood vessels that alters optical density of RBCs to interact with sunlight? Did you know that atheromatous plaques replenish the supply of cholesterol and sulfate to the microvasculature, by exploiting superoxide to derive sulfate from homocysteine and other sulfur sources?

Recall from my Ubiquitination #6 blog that in T2D superoxide pulses from cytochrome one (NAD+) is reduced or missing. If you read the thread on my forum about carotenoids you will see, in detail, how the superoxide free radical is made natural in cells via sunlight. Not all superoxide is equivalent because there is a dangerous singlet version and a non dangerous triplet version. Might the balance of superoxide type be dependent upon the optical density of the light of the sun and the optical density sensed by our skin, plasma, and arterial wall to cause atherosclerosis fundamentally?

A lack of negative charge inside the artery = more arterial aging. Notice the electromagnetic fields that are normally present in arteries and RBCs as blood flows.

In my opinion, yes. It seems now others are also seeing what I see based on the cite in this post. Putting all these ideas together you get the picture below.

michael john moreau
2022-11-10 01:19:51 +0000 UTCmichael john moreau
2022-11-10 01:10:11 +0000 UTCmichael john moreau
2022-11-10 00:49:45 +0000 UTCDr. Jack Kruse
2022-11-09 15:57:53 +0000 UTCDr. Jack Kruse
2022-11-09 15:46:44 +0000 UTCDr. Jack Kruse
2022-11-09 15:34:45 +0000 UTCChris Sussmilch
2022-11-02 10:41:09 +0000 UTCShakaMoto
2022-11-01 17:40:10 +0000 UTCWarren
2022-11-01 09:28:24 +0000 UTCBrent Patrick
2022-10-30 23:25:21 +0000 UTCmichael john moreau
2022-10-30 22:44:00 +0000 UTCLaura Kissmann
2022-10-27 18:31:32 +0000 UTCDr. Jack Kruse
2022-10-27 15:21:47 +0000 UTCRobert Koagedal
2022-10-27 15:10:21 +0000 UTCLaura Kissmann
2022-10-27 13:45:57 +0000 UTCJohnny London
2022-10-27 13:42:09 +0000 UTC