Dr. Jack Kruse

HYPOXIA #23: DOES HYPOXIA DESTROY PHOTORECEPTORS?

Added 2020-09-30 19:57:29 +0000 UTC

Is nighttime and DAYTIME technology devices use to blame for the etiology of most diseases in humans? Yes, I believe it is today. WOW. That is a big statement, Uncle Jack. How and Why? Here is a recent slide from a presentation I gave to shock my audience below.

Melanopsin, like the cone photoreceptor, is a PHOTORECEPTOR TOO FOR BLUE LIGHT. ALL OPSINS in humans are bound to retinol, and when the photoreceptors are damaged it is because of the atomic changes in retinol when the weak covalent bound it broken by light out of the normal circadian cycle.

What is the take home? The blue light hazard associated with man made light or the light present in all laboratories globally today generates massive levels of mitochondrial ROS and oxidative stress occurs directly in the outer segments of photoreceptors after blue light irradiation leading to disease. The cites below show that this statement is not hyperbole. It is now PEER reviewed published reality.

If you want to know where melanoma begins read the last cite in listed below. Melanocytes sense blue light and regulate pigmentation through Opsin-3 = melanopsin.

It took 90 years for “medical science " to rebut the claim that linoleic acid is essential in any human diet, a claim that has led to an incalculable mischief in terms of dietary advice and disease prevalence, In the last two centuries. How many decades will it take for the average physician or your physician to read those papers covering the nutrition debacle? How long will it take to change their advice or their practice of the healing arts?

I believe it might take several hundred years to get people to understand that blue light from man made devices is more deadly than any other stimulus man has created in the last 120 years because our focus in in nuclear DNA and not on the mitochondrial DNA mutations cause by blue light.

In 1998, we found melanopsin in the retina. In 2009 we found neuropsin in the skin and cornea. In 2014, we found melanopsin in the arterioles of the human body. This tells us all we are creatures of light who capture and transform the light of the sun into energy that cells use to create life and health.

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In December 2017, we found melanopsin in the skin and subcutaneous fat of humans. This was huge news to those who understand leptin and that leptin the fat hormone is also found within the subcutaneous fat of MAN. The data is telling us why we have an obesity crisis and it has NOTHING to do with food or exercise but it has a lot to do with circadian arrhythmia of light in our eye and skin and subcutaneous fat mass.

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The nighttime and daytime light environment has changed dramatically due to modern technology. Increased usage of mobile devices during ANYTIME OF THE DAY can disrupt your circadian clock. PEOPLE forget that the melanopsin receptor is regenerated DURING daytime!!!! So if you are around man-made blue light during the day you are still ruining your melanopsin photoreceptors. The intense light emitted from technology devices in screens has the potential to alter the timed release of factors within the eye, leading to chronic insults and susceptibility for visual damage. What does this mean to melanopsin photoreceptors?

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I gave you my answer in the Vermont 2018 video, and I strongly suggest you view it on Patreon here.

Recent findings cited below to suggest a functional role for the circadian clock in mammalian cone photoreceptor development and provide evidence for a continuing role for thyroid hormone (TH) signaling in cone photoreceptor maintenance. These researchers have said their findings may be of relevance in OTHER tissues where human photoreceptors are as well, where the circadian clock could alter tissue function by directly regulating enzyme type 2 iodothyronine deiodinase (Dio2) expression and thus TH signaling.

THAT is WHAT the data I presented in VERMONT 2018 is all about. I hope you view the talk. It is well worth it to further your education. Once you realize and know where melanopsin is, and follow the damage in its wake, you begin to see where mitochondrial disease begins for the very first time in your life. It happens where clinicians least expect it and that is why they always miss it and the public cannot be helped via a prescription pad. That is where the data is now........it is not in food/exercise choices, it is made in the light choices we make.

With this new information, researchers can begin to ask questions such as, “How else can we change the photoreceptors in humans using light evolution has never used? Are there other factors that can improve photoreceptor function and help extend their viability that we have failed to consider in science and industry? The BLACK SWAN among you now knows this answer. It should be as clear as the nose on your face, because this new data is telling you disease generation is not what your doctors were taught in medical school. Your light choices form your medical destiny.