THE MPEMBA EFFECT AND MELANOPSIN

Why are physicians clueless about medicine?  They have no idea how hydrogen bonding works in the hydrated proteins coded for in DNA/RNA.  If they learned how to make ice cream they would learn very quickly how hydrogen bonds also explain the Mpemba effect.  Might this be the key to understanding how to make DDW and measuring it in humans?  

I believe it will be. 

The Mpemba effect is the observation that WARM water freezes more quickly than cold water.

Water may be one of the most abundant compounds on Earth, but it is also one of more mysterious. For example, like most liquids, it becomes denser as it cools. But unlike them, it reaches a state of maximum density at 4°C and then becomes less dense before it freezes.

In solid form, it is less dense still, which is why standard ice floats on water. That’s one reason why life on Earth has flourished— if ice were denser than water, lakes, and oceans would freeze from the bottom up, almost certainly preventing the kind of chemistry that makes life possible.

Hydrogen bonds are weaker than covalent bonds but stronger than the van der Waals forces that geckos use to climb walls.

Chemists have long known that they are important. For example, water’s boiling point is much higher than other liquids of similar molecules because hydrogen bonds hold it together.

But in recent years, chemists have become increasingly aware of more subtle roles that hydrogen bonds can play but have been unable to pin down exactly why the Mpemba effect exists in water.  For example, water molecules inside narrow capillaries form into chains held together by hydrogen bonds. This plays an important role in trees and plants where water evaporation across a leaf membrane effectively pulls a chain of water molecules up from the roots.

The effect is greatest in the arterioles and capillaries of your skin and subcutaneous fat where leptin and melanopsin reside and where nitric oxide is made to alter your microcirculation and blood pressure. 

Blood is 93% liquid water by volume.  Mitochondrial make all their cell water at cytochrome C oxidase (CCO), which is the 4th cytochrome in our mitochondria. 

In liquid water, hydrogen bonds bring water molecules into close contact and when this happens anywhere in NATURE, the natural repulsion between the molecules causes the covalent O-H bonds to stretch and store energy from the environment or system where water exists.  This is really important in the skin where the sun affects the surface or in mitochondria where water is made at CCO and heat is liberated.  

But as the liquid warms up, it forces the hydrogen bonds to stretch further apart and the water molecules sit further apart. This allows the covalent molecules to shrink again and give up their energy to the environment. The important point is that this process in which the covalent bonds give up energy is equivalent to cooling.  This has massive implications when you begin to realize that the bond between melanopsin and Vitamin A is a weak covalent bond in diurnal mammals and much stronger in nocturnal mammals.  

It is clear that Nature uses the Mpemba effect between the day and night cycle.  I wonder when people will finally see the effect of nnEMF energies to this science and how it generations melanopsin dysfunction and leptin resistance?  Leptin is also a photoreceptor protein in our subcutaneous fat that is also destroyed and disrupted by melanopsin dysfunction of blue light and nnEMF.

CITES:  

Ref: arxiv.org/abs/1310.6514: O: H-O Bond Anomalous Relaxation Resolving Mpemba Paradox

J. R. Errington and P. G. Debenedetti, Relationship between structural order and the anomalies of liquid water, Nature, 409 (2001) 318-321.   (reference 169 from picture in the blog)