CPC #34:DOES RETINOL IN BLUE LIGHT or nnEMF BECOMES THE AGENT OF DOOM TO HUMAN PHOTORECEPTORS?
Added 2018-09-28 13:09:52 +0000 UTCIs nighttime and DAYTIME technology device use to blame for the etiology of most diseases in humans? Yes, it is. WOW. That is a big statement. How and Why? Here is a recent slide from a presentation I gave to shock my audience below.
Melanopsin, like the cone photoreceptor, is a PHOTORECEPTOR TOO FOR BLUE LIGHT. ALL OPSINS in humans are bound to retinol, and when the photoreceptors are damaged it is because of the atomic changes in retinol when the weak covalent bound it broken by light out of the normal circadian cycle.
In 1998, we found melanopsin in the retina. In 2014, we found melanopsin in the arterioles of the human body.
In December 2017, we found melanopsin in the skin and subcutaneous fat of humans. This was huge news to those who understand leptin and that leptin the fat hormone is also found within the subcutaneous fat of MAN. The data is telling us why we have an obesity crisis and it has NOTHING to do with food or exercise but it has a lot to do with circadian arhthmia of light in our eye and skin and subcutaneous fat mass.
The nighttime and daytime light environment has changed dramatically due to modern technology. Increased usage of mobile devices during ANYTIME OF THE DAY can disrupt your circadian clock. PEOPLE forget that the melanopsin receptor is regenerated DURING daytime!!!! So if you are around man-made blue light during the day you are still ruining your melanopsin photoreceptors. The intense light emitted from technology devices in screens has the potential to alter the timed release of factors within the eye, leading to chronic insults and susceptibility for visual damage. What does this mean to melanopsin photoreceptors?
I gave you my answer in the Vermont 2018 video, and I's strongly suggest you view it.
Recent findings cited below suggest a functional role for the circadian clock in mammalian cone photoreceptor development and provide evidence for a continuing role for thyroid hormone (TH) signaling in cone photoreceptor maintenance. These researchers have said their findings may be of relevance in OTHER tissues where human photoreceptors are as well, where the circadian clock could alter tissue function by directly regulating enzyme type 2 iodothyronine deiodinase (Dio2) expression and thus TH signaling.
THAT is WHAT the data I presented in VERMONT 2018 is all about. Once you realize and know where melanopsin is, and follow the damage in its wake, you begin to see where mitochondrial disease begins for the very first time in your life. That is where the data is now........it is not in food/exercise choices.
With this new information, researchers can begin to ask questions such as, “How else can we change the photoreceptors in humans using light evolution has never used? Are there other factors that can improve photoreceptor function and help extend their viability that we have failed to consider in science and industry? The BLACK SWAN among you now know this answer. It is as clear as the nose on your face.
CITES:
https://consultqd.clevelandclinic.org/circadian-rhythms-thyroid-hormone-and-vision-loss/
Comments
Well Jack mentions 26% blue light in summer, and 13% winter, so my guess is as there is less blue light and temperature is lower, the melanopsin/retinol bond is not broken as easily, hence less PVN stimulus and less hormones. Less hormone would be required generally because of increased binding affinity on receptors since they are depleted of deuterium in cold (due to uncoupling/heat release which favors H+ bonding). I'm seeing the connections now.
Kris Domitrovits
2018-10-02 15:10:17 +0000 UTCI guess as the seasons change, so does the color temp...so does the AM hormone release!
christiangroth
2018-10-01 17:43:25 +0000 UTCKris i think you re right! But I think it’s more due to lowered deuterium levels that increase the hormone receptor sensitivity . So cold, switching to keto diet... uncoupling Mitochondria...make heat( IR) instead of ATP...powers EZ...exclude Deuterium over H+...TCA and so on, but also increased hormone receptor sensitivity!
christiangroth
2018-10-01 17:42:26 +0000 UTCSo the only time nature allows this bond to be broken is early AM when there is blue balanced by red? Is that what stimulates the PVN and turns on the pituitary gland, which is then shut off by the diurnal UV? Can cold strengthen the retinol/melanopsin bond lowering the firing rate of the PVN, and hence why cold can lower hormones similarly to UV light? It would make sense that the cold would increase bond strength to limit stress response and favor regeneration in that environment, especially as UV goes away in northern regions.
Kris Domitrovits
2018-10-01 12:17:07 +0000 UTC
