CPC#70: MELANIN DEGRADATION OF THE CARDIAC TISSUE

Supraventricular tachycardia SVT is a broad term for a number of tachyarrhythmias that originate above the ventricular electrical conduction system (Purkinje fibers).  read the link below to learn about SVT.  This rhythm is usually the first symptom in melanin degradation of the cardiac plexus.  I showed you this picture in Quantum Engineering #47 for the first time.  

https://www.ekgstripsearch.com/SVT.htm

In ACLS codes we use adenosine via IV push to rid the heart of this detrimental rhythm when the patient is not symptomatic.  SVT can lead to rapid cardiac standstill.  We are seeing this more commonly now due to the spike protein inflammation of the heart by mRNA vaccines.  I believe this was behind the demise of Demar Hamlin and JJ Watt.  

IS THERE NOW EVIDENCE THAT INTENSE RED LIGHT can do THE same thing using the PER circadian gene?  Does this imply that red light is a drug equivalent?

YES, it does.  The picture above shows you this. 

Is there more to this circadian story you need to know?  YES.

You know my answer and you should know what Dr. Tina Kuru says about this...........Red light is a drug equivalent to adenosine use.  This is a message Big Pharma wants buried from centralized MDs.  

In this paper below in an effort to find out why red intense light can do this, researchers developed a photonic strategy using optogenetics to protect the heart using intense light to target and manipulate the function of the PER2 gene which is expressed in a circadian pattern in the part of the brain that controls circadian rhythms.  (Sounds like something Dr. Kruse would suggest no?)

You do know that sunlight is made of 42% intense IR-A light huh?

By amplifying this gene to improve the periodicity of the cardiac clocks, the researchers were just using LIGHT PHOTONS to do this job; they found that it protected cardiovascular tissues against LOW OXYGEN conditions like myocardial ischemia, caused by reduced oxygen flow to the heart.  Dr. Kruse called low oxygen situations pseudohypoxia (low NAD+).  These are all associated with low NAD+ levels in cytochrome 1 and leptin resistance with low delta psi on the inner mitochondrial membrane = low redox power.

They also discovered that the light increased cardiac ADENOSINE, a chemical that plays a role in blood flow regulation.   Hey didn't Dr. Kruse just do a massive post on ADENOSINE last week on his page?

Hey, isn't leptin resistance a synonym for melanopsin dysfunction?  Isn't melanopsin dysfunction a synonym for a melanin problem?  Yes, it is. Tell me again how that works Dr. Kruse?

Dr. Kruse Response:  Blue light and nnEMF liberate Vitamin A from our cells and cell membranes to raise its presence in the blood plasma and this lowers plasma levels of Vitamin C and Vitamin D.  This destroys melanin in the cardiac plexus and it degrades into adrenaline and noradrenaline that begins to stimulate the heart adrenergically.  It also has effects on the glylympaphatic system in the brain and on slow wave sleep.  When Vitamin A is liberated by non terrestrial light or trauma, we are inducing a light stress and this causes Vitamin A to becomes an aldehyde that becomes a wrecking ball for many non visual photoreceptors like melatonin, dopamine, NO, adrenaline, noradrenalin, L-Dopa, P-450 enzymes, RBCs, cytochromes, and melanin.

This wrecking ball action destroys the small molecule modulators of the mammalian circadian mechanism.  PER1 and PER2 are gears in that eye clock mechanism.  Once the molecular clock in the eye and peripheral clocks goes awry the implications for many neolithic diseases spiral out of control.  What are some of the Vitamin A proteins involved in this downward spiral?  

They are called retinoic acid receptor-related orphan nuclear receptors or RORs for short.  The RORs have several isoforms too called RORα-γ.  These proteins are also under the transcriptional control of CLOCK/BMAL1 heterodimers.  CLOCK and BMAL1 are positive regulators of circadian gene expression, and PER and CRY are the NEGATIVE FEEDBACK LOOP regulators that operate under day and night cycles.   

These are the positive and negative feedback arms of the circadian mechanism.  They must be coupled properly to terrestrial light to operate well and control all growth and metabolism, protein synthesis and hormone production and release.  It also controls receptor biology.  It controls EVERYTHING.  These are post transcriptional explosions that blow up the whole system of optical signaling and require the cell and mitochondrial to rebuild them all while our power plants are experiencing a brown out.  This is not ideal. 

 If they are not properly coupled to the light and dark cycles the eventual results is the extinction of both sides of the feedback loop.  That is how all human disease begins.  It is circadian biology that couples all the molecular clock genes in humans and the SCN of the eye drives the program and the major timekeeper.

Remember this lesson from the past:

Classic Paroxysmal SVT has a narrow QRS complex & has a very regular rhythm. ... A rapid heart rate will significantly reduce the time which the ventricles have to fill. This is why degradation of melanin to adrenalin and noradrenalin is a real problem.  Carefully look at how melanin degrades on the top line during periods of hypoxia.  The chemistry in hypoxia goes from right to left.  

The heart fills during diastole, and diastole is normally 2/3 the cardiac cycle. A rapid heart rate will significantly reduce the time which the ventricles have to fill and as a result this lowers cardiac output and oxygen delivery = hypoxia.  As a result of this the heart tries to beat faster to improve global hypoxia and this worsens the situation.  It fast forwards on itself.  The reduced filling time results in a smaller amount of blood ejected from the heart during systole. The end result is a drop in cardiac output & hypotension.

With the drop in cardiac output, a patient may experience the following symptoms. These symptoms occur more frequently with a heart rate >150 beats per minute as the ECG strip shows:

Shortness of air (S)

Palpitation feeling in the chest (S)

Ongoing chest pain (U)

Dizziness (S)

Rapid breathing (S)

Loss of consciousness (U)

Numbness of body parts (S)

The pathway of choice for SVT in the tachycardia algorithm is based on whether the patient is stable or unstable clinically.

The symptoms listed above that would indicate the patient is unstable are noted with the letter (U) in a code situation. This can present outside a code situation when someone has a very low redox state because of a very poor environment linked to blue light and nnEMF toxicity. This mimics adrenal fatigue and brainstem pathology, sleep disorders, and eating disorders I wrote about in the QE#47 blog.  Stable but serious symptoms are indicated with the letter (S) above.

Insert any 3G-5G city or environment = an acute or chronic adenosine problem. What fucks up sleep ultimately at the dorsal longitudinal fasiculus? Problems with adenosine at the brain stem level. This is why ACLS algorithms for advanced caridiac life support uses adenosine to treat acute mitochondrial failure in the heart that results in SVT cardiac rhythms........guess what drug is used for cardiac tachycardias in ACLS?

ADENOSINE via IV push = a DEFECT IN PER 1 or PER2.  SOUND FAMILIAR?

How do you like me now............?

Do you still think tech use and abuse is safe and has no side negative effects for the heart of your blood vessels?  That damage can be found early in your brainstem by the DLF where glylymphatic drainage occurs.  How about your eye clock or the molecular clocks in your skin.  These are all places where melanin, melatonin, melanopsin and leptin are.

Adenosine building up occurs normally during the day when red light is powerful as we live and is one thing that drives us to feel sleepy during the night. Then while you sleep, the adenosine is cleared out by the glylymphatic system in the brainstem at the perivascular spaces, leaving you refreshed and ready to go in the morning… for most people. This does not happen in those with sleep apnea.  Those with OSA have circadian mediated glylymphatic failure of AQA 4 in the brain stem due to lowered periodicity of PER1 & PER2 (above pic).  I can see the widened perivascular spaces on 3 Tesla MRI images at the DLF.  This is how decentralized medicine is practiced.  Today, most people that have a version of EHS that affects adenosine at the brainstem level which causes their sleep destruction and hypoxia via this mechanism. This mimics adrenal problems (PVN) and causes wild symptoms doctors cannot explain.

Who else can get these problems sans a technology addiction?

if you follow your genetics from 23 and me...

There is a genetic variant in the adenosine deaminase (ADA) gene that decreases the clearance rate of adenosine. People who carry the variant may get more slow-wave, deep sleep at night, but they also may need to sleep a little longer o feel refreshed the next morning.  These people are at high risk of neurodegeneration and for normal pressure hydrocephalus and glylymphatic failure.

What is the decentralized diagnosis that your centralized sleep MDs will never resolve for you?

Check your genetic data for rs73598374 (23andMe v4, v5; AncestryDNA):

C/C: normal clearance of adenosine

C/T: reduced clearance of adenosine, more deep sleep but may feel sleepy when waking up

T/T: reduce clearance of adenosine, more deep sleep but may feel sleepy when waking up.

How do you like me now?

Intense RED LIGHT via PBM/LLLT is the first step I like I use at Kruse Longevity Center for melanin renovation of the cardiac plexus that I wrote about in QE #47.  It is always cardioprotective.  The red light of the sun is better than my fake red light panels.  Shocking huh?

It shocks only those who are not on the path of becoming a decentralized Black Swan Mitochondriac.

If you do not listen to me you might wind up needing a shock from a defribrillator sometime in your life from tech abuse or from excessive spike protein build up in and around these areas in your body.  See my blog on Demar Hamlin or JJ Watt for more detail.

When you know better you do better.

CITES

https://www.sciencedaily.com/releases/2019/08/190808115052.htm